Aging: Driven by a few causes or many?

Link to the preprint: https://www.biorxiv.org/content/10.1101/2025.04.22.649947v1 Key-words: Actuarial aging rate, Adult mice, Age-related pathologies, Aging, Aging rate, Anti-aging interventions, Atherosclerosis, Basal vulnerability, Biological processes, Cancer risk, Cellular senescence, Core aging processes, Cross-talk, Damage accumulation, Deceleration of aging, Differential equations, Evolutionary adjustments, Exponential increase in mortality, Feedback loops, Functional decline, Gene therapy, Genetic architecture, Genetic variation, Genomic instability, Geroscience, Glucose tolerance, Gompertz mortality model, Gompertz slope parameter, Hallmarks of aging, Healthy lifespan, Human life expectancy, Immune system, Interdependent processes, Lifespans, Line of least resistance, Longevity, Mammalian model, Mathematical modelling, Mechanisms of ageing, Modifiability of aging, Mortality rate, Muscle strength, Novel interventions, Physical performance, Positive feedback, Public health, Radical life extension, Senescent cells, Statistical significance, Survival data